Publikationen in Zusammenarbeit mit Forschern von Instituto de Investigación Biomédica de Lleida (10)

2023

  1. NOD mouse dorsal root ganglia display morphological and gene expression defects before and during autoimmune diabetes development

    Frontiers in Endocrinology, Vol. 14

  2. Persistent NRG1 Type III Overexpression in Spinal Motor Neurons Has No Therapeutic Effect on ALS-Related Pathology in SOD1G93A Mice

    Neurotherapeutics, Vol. 20, Núm. 6, pp. 1820-1834

2022

  1. Accumulation of misfolded SOD1 outlines distinct patterns of motor neuron pathology and death during disease progression in a SOD1G93A mouse model of amyotrophic lateral sclerosis

    Brain Pathology, Vol. 32, Núm. 6

2021

  1. Microglial recruitment and mechanisms involved in the disruption of afferent synaptic terminals on spinal cord motor neurons after acute peripheral nerve injury

    GLIA, Vol. 69, Núm. 5, pp. 1216-1240

2018

  1. Glial activation and central synapse loss, but not motoneuron degeneration, are prevented by the sigma-1 receptor agonist pre-084 in the SMN2B/ mouse model of spinal muscular atrophy

    Journal of Neuropathology and Experimental Neurology, Vol. 77, Núm. 7, pp. 577-597

2014

  1. Accumulation of misfolded SOD1 in dorsal root ganglion degenerating proprioceptive sensory neurons of transgenic mice with amyotrophic lateral sclerosis

    BioMed Research International, Vol. 2014

  2. Neuregulin-1 is concentrated in the postsynaptic subsurface cistern of C-bouton inputs to α-motoneurons and altered during motoneuron diseases

    FASEB Journal, Vol. 28, Núm. 8, pp. 3618-3632

2013

  1. Dyrk1A Is Dynamically Expressed on Subsets of Motor Neurons and in the Neuromuscular Junction: Possible Role in Down Syndrome

    PLoS ONE, Vol. 8, Núm. 1

2011

  1. Defective neuromuscular junction organization and postnatal myogenesis in mice with severe spinal muscular atrophy

    Journal of Neuropathology and Experimental Neurology, Vol. 70, Núm. 6, pp. 444-461

2007

  1. Excitotoxic motoneuron disease in chick embryo evolves with autophagic neurodegeneration and deregulation of neuromuscular innervation

    Journal of Neuroscience Research