- Medicina Experimental
Departament
![Foto de Jordi](/img/nophoto.png)
Jordi
Bermúdez Mas
Publikationen, an denen er mitarbeitet Jordi Bermúdez Mas (10)
2009
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Fructose 1,6-bisphosphate reduced TNF-α-induced apoptosis in galactosamine sensitized rat hepatocytes through activation of nitric oxide and cGMP production
European Journal of Pharmacology, Vol. 610, Núm. 1-3, pp. 128-133
2006
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Fructose 1,6-bisphosphate prevented endotoxemia, macrophage activation, and liver injury induced by D-galactosamine in rats
Critical Care Medicine, Vol. 34, Núm. 3, pp. 807-814
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Overcoming diabetes-induced hyperglycemia through inhibition of hepatic phosphoenolpyruvate carboxykinase (GTP) with RNAi
Molecular Therapy, Vol. 13, Núm. 2, pp. 401-410
2005
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Aspirin inhibits NF-κB activation in a glycolysis-depleted lung epithelial cell line
European Journal of Pharmacology, Vol. 517, Núm. 3, pp. 158-164
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Gender dimorphism in rat liver mitochondrial oxidative metabolism and biogenesis
American Journal of Physiology - Cell Physiology, Vol. 289, Núm. 2 58-2
2004
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Glutathione content and adaptation to endogenously induced energy depletion in Mv1Lu cells
Free Radical Biology and Medicine, Vol. 36, Núm. 12, pp. 1555-1565
2002
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Enhanced antioxidant defenses and resistance to TNF-α in a glycolysis-depleted lung epithelial cell line
Free Radical Biology and Medicine, Vol. 33, Núm. 10, pp. 1409-1418
2000
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Cells overexpressing fructose-2,6-bisphosphatase showed enhanced pentose phosphate pathway flux and resistance to oxidative stress
FEBS Letters, Vol. 480, Núm. 2-3, pp. 261-264
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MPP+-induced mitochondrial dysfunction is potentiated by dopamine
Biochemical and Biophysical Research Communications, Vol. 268, Núm. 3, pp. 916-920
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Overexpression of fructose 2,6-bisphosphatase decreases glycolysis and delays cell cycle progression
American Journal of Physiology - Cell Physiology, Vol. 279, Núm. 5 48-5