- Ciències Mèdiques Bàsiques
Departament
Centro de Investigación Biomédica en Red sobre Enfermedades Neurodegenerativas
Madrid, EspañaPublicacións en colaboración con investigadores/as de Centro de Investigación Biomédica en Red sobre Enfermedades Neurodegenerativas (12)
2024
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An Insertion Within SIRPβ1 Shows a Dual Effect Over Alzheimer's Disease Cognitive Decline Altering the Microglial Response
Journal of Alzheimer's Disease, Vol. 98, Núm. 2, pp. 601-618
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Assessment of the Correlation and Diagnostic Accuracy between Cerebrospinal Fluid and Plasma Alzheimer’s Disease Biomarkers: A Comparison of the Lumipulse and Simoa Platforms
International Journal of Molecular Sciences, Vol. 25, Núm. 9
2021
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Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)1
Autophagy, Vol. 17, Núm. 1, pp. 1-382
2020
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SIVA-1 regulates apoptosis and synaptic function by modulating XIAP interaction with the death receptor antagonist FAIM-L
Cell Death and Disease, Vol. 11, Núm. 2
2019
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Genome Wide Meta-Analysis identifies common genetic signatures shared by heart function and Alzheimer’s disease
Scientific Reports, Vol. 9, Núm. 1
2018
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Lipid Profile in Human Frontal Cortex Is Sustained Throughout Healthy Adult Life Span to Decay at Advanced Ages
Journals of Gerontology - Series A Biological Sciences and Medical Sciences, Vol. 73, Núm. 6, pp. 703-710
2017
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Region-specific vulnerability to lipid peroxidation and evidence of neuronal mechanisms for polyunsaturated fatty acid biosynthesis in the healthy adult human central nervous system
Biochimica et Biophysica Acta - Molecular and Cell Biology of Lipids, Vol. 1862, Núm. 5, pp. 485-495
2013
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FAIM-L is an IAP-binding protein that inhibits XIAP ubiquitinylation and protects from fas-induced apoptosis
Journal of Neuroscience, Vol. 33, Núm. 49, pp. 19262-19275
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Neurobehavioral characterization of Endonuclease G knockout mice reveals a new putative molecular player in the regulation of anxiety
Experimental Neurology, Vol. 247, pp. 122-129
2011
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Endonuclease G is a novel determinant of cardiac hypertrophy and mitochondrial function
Nature, Vol. 478, Núm. 7367, pp. 114-118
2010
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The death receptor antagonist FLIP-L interacts with Trk and is necessary for neurite outgrowth induced by neurotrophins
Journal of Neuroscience, Vol. 30, Núm. 17, pp. 6094-6105
2008
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BCL-X L regulates TNF-α-mediated cell death independently of NF-kB, FLIP and IAPs
Cell Research, Vol. 18, Núm. 10, pp. 1020-1036