Publicaciones en las que colabora con MARIO ENCINAS MARTIN (15)

2024

  1. Sprouty1 is a broad mediator of cellular senescence

    Cell Death and Disease, Vol. 15, Núm. 4

2023

  1. In Vivo Intra-Uterine Delivery of TAT-Fused Cre Recombinase and CRISPR/Cas9 Editing System in Mice Unveil Histopathology of Pten/p53-Deficient Endometrial Cancers

    Advanced Science, Vol. 10, Núm. 32

  2. Metformin exhibits antineoplastic effects on Pten-deficient endometrial cancer by interfering with TGF-β and p38/ERK MAPK signalling

    Biomedicine and Pharmacotherapy, Vol. 168

  3. Transient and DNA-free in vivo CRISPR/Cas9 genome editing for flexible modeling of endometrial carcinogenesis

    Cancer Communications

2022

  1. A dominant negative mutation uncovers cooperative control of caudal Wolffian duct development by Sprouty genes

    Cellular and Molecular Life Sciences, Vol. 79, Núm. 10

  2. Lack of extracellular matrix switches TGF-β induced apoptosis of endometrial cells to epithelial to mesenchymal transition

    Scientific Reports, Vol. 12, Núm. 1

2021

  1. Endometrial pten deficiency leads to smad2/3 nuclear translocation

    Cancers, Vol. 13, Núm. 19

2019

  1. Barley β-glucan accelerates wound healing by favoring migration versus proliferation of human dermal fibroblasts

    Carbohydrate Polymers, Vol. 210, pp. 389-398

  2. Sprouty1 controls genitourinary development via its N-terminal tyrosine

    Journal of the American Society of Nephrology, Vol. 30, Núm. 8, pp. 1398-1411

2017

  1. Palbociclib has antitumour effects on Pten-deficient endometrial neoplasias

    Journal of Pathology, Vol. 242, Núm. 2, pp. 152-164

2009

  1. RANKL increases vascular smooth muscle cell calcification through a rank-bmp4-dependent pathway

    Circulation Research, Vol. 104, Núm. 9, pp. 1041-1048

2006

  1. Pro-NGF from Alzheimer's disease and normal human brain displays distinctive abilities to induce processing and nuclear translocation of intracellular domain of p75NTR and apoptosis

    American Journal of Pathology, Vol. 169, Núm. 1, pp. 119-131

2004

  1. Trk is a calmodulin-binding protein: Implications for receptor processing

    Journal of Neurochemistry, Vol. 88, Núm. 2, pp. 422-433

2001

  1. Neuronal survival induced by neurotrophins requires calmodulin

    Journal of Cell Biology, Vol. 154, Núm. 3, pp. 585-597

1999

  1. Receptors of the glial cell line-derived neurotrophic factor family of neurotrophic factors signal cell survival through the phosphatidylinositol 3- kinase pathway in spinal cord motoneurons

    Journal of Neuroscience, Vol. 19, Núm. 21, pp. 9160-9169