Publicaciones (24) Publicaciones en las que ha participado algún/a investigador/a

2014

  1. Calpain activation and CaMKIV reduction in spinal cords from hSOD1G93A mouse model

    Molecular and Cellular Neuroscience, Vol. 61, pp. 219-225

  2. Evaluación ecológica en el deterioro cognitivo leve y enfermedad de Alzheimer mediante el Rivermead Behavioural Memory Test

    Neurologia, Vol. 29, Núm. 6, pp. 339-345

  3. N-terminal pro-brain natriuretic peptide level determined at different times identifies transient ischaemic attack patients with atrial fibrillation

    European Journal of Neurology, Vol. 21, Núm. 4, pp. 679-683

  4. Sprouty1 induces a senescence-associated secretory phenotype by regulating NFκB activity: Implications for tumorigenesis

    Cell Death and Differentiation, Vol. 21, Núm. 2, pp. 333-343

2013

  1. Differential gene expression of medullary thyroid carcinoma reveals specific markers associated with genetic conditions

    American Journal of Pathology, Vol. 182, Núm. 2, pp. 350-362

  2. NF-κB signaling pathways: Role in nervous system physiology and pathology

    Neuroscientist, Vol. 19, Núm. 2, pp. 175-194

  3. Notch signaling pathway is activated in motoneurons of spinal muscular atrophy

    International Journal of Molecular Sciences, Vol. 14, Núm. 6, pp. 11424-11437

  4. Survival motor neuron protein reduction deregulates autophagy in spinal cord motoneurons in vitro.

    Cell death & disease, Vol. 4

2012

  1. SMN deficiency attenuates migration of U87MG astroglioma cells through the activation of RhoA

    Molecular and Cellular Neuroscience, Vol. 49, Núm. 3, pp. 282-289

  2. Sprouty1 is a candidate tumor-suppressor gene in medullary thyroid carcinoma

    Oncogene, Vol. 31, Núm. 35, pp. 3961-3972

2011

  1. A new model to study spinal muscular atrophy: Neurite degeneration and cell death is counteracted by BCL-XL Overexpression in motoneurons

    Neurobiology of Disease, Vol. 42, Núm. 3, pp. 415-426

  2. KSR1 is overexpressed in endometrial carcinoma and regulates proliferation and TRAIL-induced apoptosis by modulating FLIP levels

    American Journal of Pathology, Vol. 178, Núm. 4, pp. 1529-1543

  3. Nuclear factor-B2/p100 promotes endometrial carcinoma cell survival under hypoxia in a HIF-1α independent manner

    Laboratory Investigation, Vol. 91, Núm. 6, pp. 859-871

  4. Promoter hypermethylation and expression of sprouty 2 in endometrial carcinoma

    Human Pathology, Vol. 42, Núm. 2, pp. 185-193

  5. The canonical nuclear factor-κB pathway regulates cell survival in a developmental model of spinal cord motoneurons

    Journal of Neuroscience, Vol. 31, Núm. 17, pp. 6493-6503

2010

  1. Study of the in vivo phosphorylation of E2F1 on Ser403

    Biochimica et Biophysica Acta - Molecular Cell Research, Vol. 1803, Núm. 8, pp. 912-918

  2. The death receptor antagonist FLIP-L interacts with Trk and is necessary for neurite outgrowth induced by neurotrophins

    Journal of Neuroscience, Vol. 30, Núm. 17, pp. 6094-6105

2009

  1. 1,25-Dihydroxyvitamin D3 regulates VEGF production through a vitamin D response element in the VEGF promoter

    Atherosclerosis, Vol. 204, Núm. 1, pp. 85-89

  2. Cell death and learning impairment in mice caused by in vitro modified pro-NGF can be related to its increased oxidative modifications in Alzheimer disease

    American Journal of Pathology, Vol. 175, Núm. 6, pp. 2574-2585

  3. Loss of Sprouty1 rescues renal agenesis caused by ret mutation

    Journal of the American Society of Nephrology, Vol. 20, Núm. 2, pp. 255-259