Publicaciones en las que colabora con Josep E. Esquerda Colell (33)

2023

  1. Persistent NRG1 Type III Overexpression in Spinal Motor Neurons Has No Therapeutic Effect on ALS-Related Pathology in SOD1G93A Mice

    Neurotherapeutics, Vol. 20, Núm. 6, pp. 1820-1834

2022

  1. Accumulation of misfolded SOD1 outlines distinct patterns of motor neuron pathology and death during disease progression in a SOD1G93A mouse model of amyotrophic lateral sclerosis

    Brain Pathology, Vol. 32, Núm. 6

2021

  1. Beneficial effects of dietary supplementation with green tea catechins and cocoa flavanols on aging-related regressive changes in the mouse neuromuscular system

    Aging, Vol. 13, Núm. 14, pp. 18051-18093

  2. Microglial recruitment and mechanisms involved in the disruption of afferent synaptic terminals on spinal cord motor neurons after acute peripheral nerve injury

    GLIA, Vol. 69, Núm. 5, pp. 1216-1240

2020

  1. Motoneuron deafferentation and gliosis occur in association with neuromuscular regressive changes during ageing in mice

    Journal of Cachexia, Sarcopenia and Muscle, Vol. 11, Núm. 6, pp. 1628-1660

  2. The Y172 Monoclonal Antibody Against p-c-Jun (Ser63) Is a Marker of the Postsynaptic Compartment of C-Type Cholinergic Afferent Synapses on Motoneurons

    Frontiers in Cellular Neuroscience, Vol. 13

2019

  1. Localization and dynamic changes of neuregulin-1 at C-type synaptic boutons in association with motor neuron injury and repair

    FASEB Journal, Vol. 33, Núm. 7, pp. 7833-7851

2018

  1. Glial activation and central synapse loss, but not motoneuron degeneration, are prevented by the sigma-1 receptor agonist pre-084 in the SMN2B/ mouse model of spinal muscular atrophy

    Journal of Neuropathology and Experimental Neurology, Vol. 77, Núm. 7, pp. 577-597

2017

  1. Neuregulin 1-ErbB module in C-bouton synapses on somatic motor neurons: Molecular compartmentation and response to peripheral nerve injury

    Scientific Reports, Vol. 7

2016

  1. Chronic Treatment with the AMPK Agonist AICAR Prevents Skeletal Muscle Pathology but Fails to Improve Clinical Outcome in a Mouse Model of Severe Spinal Muscular Atrophy

    Neurotherapeutics, Vol. 13, Núm. 1, pp. 198-216

2014

  1. Accumulation of misfolded SOD1 in dorsal root ganglion degenerating proprioceptive sensory neurons of transgenic mice with amyotrophic lateral sclerosis

    BioMed Research International, Vol. 2014

  2. Mechanisms involved in spinal cord central synapse loss in a mouse model of spinal muscular atrophy

    Journal of Neuropathology and Experimental Neurology, Vol. 73, Núm. 6, pp. 519-535

  3. Neuregulin-1 is concentrated in the postsynaptic subsurface cistern of C-bouton inputs to α-motoneurons and altered during motoneuron diseases

    FASEB Journal, Vol. 28, Núm. 8, pp. 3618-3632

2013

  1. Chronic treatment with lithium does not improve neuromuscular phenotype in a mouse model of severe spinal muscular atrophy

    Neuroscience, Vol. 250, pp. 417-433

2011

  1. Defective neuromuscular junction organization and postnatal myogenesis in mice with severe spinal muscular atrophy

    Journal of Neuropathology and Experimental Neurology, Vol. 70, Núm. 6, pp. 444-461

2010

  1. Increased intramuscular nerve branching and inhibition of programmed cell death of chick embryo motoneurons by immunoglobulins from patients with motoneuron disease

    Journal of Neuroimmunology, Vol. 229, Núm. 1-2, pp. 157-168

  2. Lithium prevents excitotoxic cell death of motoneurons in organotypic slice cultures of spinal cord

    Neuroscience, Vol. 165, Núm. 4, pp. 1353-1369

2009

  1. Development of microglia in the chick embryo spinal cord: Implications in the regulation of motoneuronal survival and death

    Journal of Neuroscience Research, Vol. 87, Núm. 11, pp. 2447-2466

  2. Excitotoxic motoneuron degeneration induced by glutamate receptor agonists and mitochondrial toxins in organotypic cultures of chick embryo spinal cord

    Journal of Comparative Neurology, Vol. 516, Núm. 4, pp. 277-290

2008

  1. The rescue of developing avian motoneurons from programmed cell death by a selective inhibitor of the fetal muscle-specific nicotinic acetylcholine receptor

    Developmental Neurobiology, Vol. 68, Núm. 7, pp. 972-980