![Foto de OLGA](/img/nophoto.png)
OLGA
TARABAL MOSTAZO
TU
Publicaciones (26) Publicaciones de OLGA TARABAL MOSTAZO
2023
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Persistent NRG1 Type III Overexpression in Spinal Motor Neurons Has No Therapeutic Effect on ALS-Related Pathology in SOD1G93A Mice
Neurotherapeutics, Vol. 20, Núm. 6, pp. 1820-1834
2021
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Beneficial effects of dietary supplementation with green tea catechins and cocoa flavanols on aging-related regressive changes in the mouse neuromuscular system
Aging, Vol. 13, Núm. 14, pp. 18051-18093
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Microglial recruitment and mechanisms involved in the disruption of afferent synaptic terminals on spinal cord motor neurons after acute peripheral nerve injury
GLIA, Vol. 69, Núm. 5, pp. 1216-1240
2020
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Motoneuron deafferentation and gliosis occur in association with neuromuscular regressive changes during ageing in mice
Journal of Cachexia, Sarcopenia and Muscle, Vol. 11, Núm. 6, pp. 1628-1660
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The Y172 Monoclonal Antibody Against p-c-Jun (Ser63) Is a Marker of the Postsynaptic Compartment of C-Type Cholinergic Afferent Synapses on Motoneurons
Frontiers in Cellular Neuroscience, Vol. 13
2019
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Localization and dynamic changes of neuregulin-1 at C-type synaptic boutons in association with motor neuron injury and repair
FASEB Journal, Vol. 33, Núm. 7, pp. 7833-7851
2018
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Accumulation of poly(A) RNA in nuclear granules enriched in Sam68 in motor neurons from the SMNΔ7 mouse model of SMA
Scientific Reports, Vol. 8, Núm. 1
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Glial activation and central synapse loss, but not motoneuron degeneration, are prevented by the sigma-1 receptor agonist pre-084 in the SMN2B/ mouse model of spinal muscular atrophy
Journal of Neuropathology and Experimental Neurology, Vol. 77, Núm. 7, pp. 577-597
2017
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Cellular bases of the RNA metabolism dysfunction in motor neurons of a murine model of spinal muscular atrophy: Role of Cajal bodies and the nucleolus
Neurobiology of Disease, Vol. 108, pp. 83-99
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Neuregulin 1-ErbB module in C-bouton synapses on somatic motor neurons: Molecular compartmentation and response to peripheral nerve injury
Scientific Reports, Vol. 7
2016
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Chronic Treatment with the AMPK Agonist AICAR Prevents Skeletal Muscle Pathology but Fails to Improve Clinical Outcome in a Mouse Model of Severe Spinal Muscular Atrophy
Neurotherapeutics, Vol. 13, Núm. 1, pp. 198-216
2014
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Accumulation of misfolded SOD1 in dorsal root ganglion degenerating proprioceptive sensory neurons of transgenic mice with amyotrophic lateral sclerosis
BioMed Research International, Vol. 2014
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Mechanisms involved in spinal cord central synapse loss in a mouse model of spinal muscular atrophy
Journal of Neuropathology and Experimental Neurology, Vol. 73, Núm. 6, pp. 519-535
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Neuregulin-1 is concentrated in the postsynaptic subsurface cistern of C-bouton inputs to α-motoneurons and altered during motoneuron diseases
FASEB Journal, Vol. 28, Núm. 8, pp. 3618-3632
2010
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Lithium prevents excitotoxic cell death of motoneurons in organotypic slice cultures of spinal cord
Neuroscience, Vol. 165, Núm. 4, pp. 1353-1369
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Neurotoxic species of misfolded SOD1G93A recognized by antibodies against the P2X4 subunit of the ATP receptor accumulate in damaged neurons of transgenic animal models of amyotrophic lateral sclerosis
Journal of Neuropathology and Experimental Neurology, Vol. 69, Núm. 2, pp. 176-187
2009
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Excitotoxic motoneuron degeneration induced by glutamate receptor agonists and mitochondrial toxins in organotypic cultures of chick embryo spinal cord
Journal of Comparative Neurology, Vol. 516, Núm. 4, pp. 277-290
2008
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Strong P2X4 purinergic receptor-like immunoreactivity is selectively associated with degenerating neurons in transgenic rodent models of amyotrophic lateral sclerosis
Journal of Comparative Neurology, Vol. 506, Núm. 1, pp. 75-92
2007
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Excitotoxic motoneuron disease in chick embryo evolves with autophagic neurodegeneration and deregulation of neuromuscular innervation
Journal of Neuroscience Research
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Survival and death of mature avian motoneurons in organotypic slice culture: Trophic requirements for survival and different types of degeneration
Journal of Comparative Neurology, Vol. 501, Núm. 5, pp. 669-690