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Instituto de Investigación Biomédica de Lleida
Lleida, EspañaPublicaciones en colaboración con investigadores/as de Instituto de Investigación Biomédica de Lleida (24)
2024
2023
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Oxidative stress-induced FAK activation contributes to uterine serous carcinoma aggressiveness
Molecular Oncology, Vol. 17, Núm. 1, pp. 98-118
2022
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Mice harboring the FXN I151F pathological point mutation present decreased frataxin levels, a Friedreich ataxia-like phenotype, and mitochondrial alterations
Cellular and Molecular Life Sciences, Vol. 79, Núm. 2
2021
2020
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Mitochondrial localization of the yeast forkhead factor hcm1
International Journal of Molecular Sciences, Vol. 21, Núm. 24, pp. 1-11
2019
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Loss of glutathione redox homeostasis impairs proteostasis by inhibiting autophagy-dependent protein degradation
Cell Death and Differentiation, Vol. 26, Núm. 9, pp. 1545-1565
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Redox control of yeast Sir2 activity is involved in acetic acid resistance and longevity
Redox Biology, Vol. 24
2017
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Abnormal degradation of the neuronal stress-protective transcription factor HSF1 in Huntington's disease
Nature Communications, Vol. 8
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Specificity of Protein Carbonylation and Its Relevance in Aging
Protein Carbonylation: Principles, Analysis, and Biological Implications (wiley), pp. 340-383
2016
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2-phenylethynesulphonamide (PFT-μ) enhances the anticancer effect of the novel hsp90 inhibitor NVP-AUY922 in melanoma, by reducing GSH levels
Pigment Cell and Melanoma Research, Vol. 29, Núm. 3, pp. 352-371
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Impaired PLP-dependent metabolism in brain samples from Huntington disease patients and transgenic R6/1 mice
Metabolic Brain Disease, Vol. 31, Núm. 3, pp. 579-586
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Reversible glutathionylation of Sir2 by monothiol glutaredoxins Grx3/4 regulates stress resistance
Free Radical Biology and Medicine, Vol. 96, pp. 45-56
2013
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The FOX transcription factor Hcm1 regulates oxidative metabolism in response to early nutrient limitation in yeast. Role of Snf1 and Tor1/Sch9 kinases
Biochimica et Biophysica Acta - Molecular Cell Research, Vol. 1833, Núm. 8, pp. 2004-2015
2012
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Correction: Reduction of oxidative cellular damage by overexpression of the thioredoxin TRX2 gene improves yield and quality of wine yeast dry active biomass
Microbial Cell Factories
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Engineered Trx2p industrial yeast strain protects glycolysis and fermentation proteins from oxidative carbonylation during biomass propagation
Microbial Cell Factories, Vol. 11
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Protein oxidation in Huntington disease
BioFactors, Vol. 38, Núm. 3, pp. 173-185
2011
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Sir2 is induced by oxidative stress in a yeast model of Huntington disease and its activation reduces protein aggregation
Archives of Biochemistry and Biophysics, Vol. 510, Núm. 1, pp. 27-34
2010
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Protein oxidation in Huntington disease affects energy production and vitamin B6 metabolism
Free Radical Biology and Medicine, Vol. 49, Núm. 4, pp. 612-621
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Proteomic strategies for the analysis of carbonyl groups on proteins
Current Protein and Peptide Science, Vol. 11, Núm. 8, pp. 652-658
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Reduction of oxidative cellular damage by overexpression of the thioredoxin TRX2 gene improves yield and quality of wine yeast dry active biomass
Microbial Cell Factories, Vol. 9