Publicaciones en colaboración con investigadores/as de Instituto de Investigación Biomédica de Lleida (23)

2023

  1. SIRT6 regulates obesity-induced oxidative stress via ENDOG/SOD2 signaling in the heart

    Cell Biology and Toxicology, Vol. 39, Núm. 4, pp. 1489-1507

2022

  1. Cardiac fibroblasts display endurance to ischemia, high ROS control and elevated respiration regulated by the JAK2/STAT pathway

    FEBS Journal, Vol. 289, Núm. 9, pp. 2540-2561

2021

  1. A trans locus causes a ribosomopathy in hypertrophic hearts that affects mRNA translation in a protein length-dependent fashion

    Genome Biology, Vol. 22, Núm. 1

  2. Endog impacts on tumor cell proliferation and tumor prognosis in the context of pi3k/pten pathway status

    Cancers, Vol. 13, Núm. 15

2020

  1. AKT2 regulates development and metabolic homeostasis via AMPK-depedent pathway in skeletal muscle

    Clinical Science, Vol. 134, Núm. 17, pp. 2381-2398

2019

  1. Nrf2 deficiency aggravates Angiotensin II-induced cardiac injury by increasing hypertrophy and enhancing IL-6/STAT3-dependent inflammation

    Biochimica et Biophysica Acta - Molecular Basis of Disease, Vol. 1865, Núm. 6, pp. 1253-1264

2018

  1. Cardiomyocyte hypertrophy induced by Endonuclease G deficiency requires reactive oxygen radicals accumulation and is inhibitable by the micropeptide humanin

    Redox Biology, Vol. 16, pp. 146-156

  2. Deletion of protein kinase B2 preserves cardiac function by blocking interleukin-6-mediated injury and restores blood pressure during angiotensin II/high-salt-diet-induced hypertension

    Journal of Hypertension, Vol. 36, Núm. 4, pp. 834-846

2017

  1. AKT2 blocks nucleus translocation of apoptosis-inducing factor (AIF) and endonuclease G (EndoG) while promoting caspase activation during cardiac ischemia

    International Journal of Molecular Sciences, Vol. 18, Núm. 3

  2. Interleukin-6 deficiency facilitates myocardial dysfunction during high fat diet-induced obesity by promoting lipotoxicity and inflammation

    Biochimica et Biophysica Acta - Molecular Basis of Disease, Vol. 1863, Núm. 12, pp. 3128-3141

2016

  1. EndoG knockout mice show increased Brown adipocyte recruitment in white adipose tissue and improved glucose homeostasis

    Endocrinology, Vol. 157, Núm. 10, pp. 3873-3887

  2. Studies on the role of apoptosis after transient myocardial ischemia: genetic deletion of the executioner caspases-3 and -7 does not limit infarct size and ventricular remodeling

    Basic Research in Cardiology, Vol. 111, Núm. 2, pp. 1-10

2014

  1. Histone deacetylase inhibitors promote glioma cell death by G2 checkpoint abrogation leading to mitotic catastrophe

    Cell Death and Disease, Vol. 5, Núm. 10

2013

  1. FAIM-L is an IAP-binding protein that inhibits XIAP ubiquitinylation and protects from fas-induced apoptosis

    Journal of Neuroscience, Vol. 33, Núm. 49, pp. 19262-19275

  2. Neurobehavioral characterization of Endonuclease G knockout mice reveals a new putative molecular player in the regulation of anxiety

    Experimental Neurology, Vol. 247, pp. 122-129

2012

  1. Translation of Myocyte Enhancer Factor-2 is induced by hypertrophic stimuli in cardiomyocytes through a Calcineurin-dependent pathway

    Journal of Molecular and Cellular Cardiology, Vol. 53, Núm. 4, pp. 578-587

2011

  1. Endog links Bnip3-induced mitochondrial damage and caspase-independent DNA fragmentation in ischemic cardiomyocytes

    PLoS ONE, Vol. 6, Núm. 3

  2. Endonuclease G is a novel determinant of cardiac hypertrophy and mitochondrial function

    Nature, Vol. 478, Núm. 7367, pp. 114-118

  3. Ubiquitination of TrkA by Nedd4-2 regulates receptor lysosomal targeting and mediates receptor signaling

    Journal of Neurochemistry, Vol. 117, Núm. 3, pp. 479-493

2009

  1. Polypyrimidine tract binding proteins (PTB) regulate the expression of apoptotic genes and susceptibility to caspase-dependent apoptosis in differentiating cardiomyocytes

    Cell Death and Differentiation, Vol. 16, Núm. 11, pp. 1460-1468