Publicaciones en colaboración con investigadores/as de Universitat de Barcelona (36)

2022

  1. Cardiac fibroblasts display endurance to ischemia, high ROS control and elevated respiration regulated by the JAK2/STAT pathway

    FEBS Journal, Vol. 289, Núm. 9, pp. 2540-2561

2021

  1. Endog impacts on tumor cell proliferation and tumor prognosis in the context of pi3k/pten pathway status

    Cancers, Vol. 13, Núm. 15

2020

  1. Involvement of the mitochondrial nuclease EndoG in the regulation of cell proliferation through the control of reactive oxygen species

    Redox Biology, Vol. 37

2015

  1. Executioner caspase-3 and 7 deficiency reduces myocyte number in the developing mouse heart

    PLoS ONE, Vol. 10, Núm. 6

2011

  1. Endog links Bnip3-induced mitochondrial damage and caspase-independent DNA fragmentation in ischemic cardiomyocytes

    PLoS ONE, Vol. 6, Núm. 3

2010

  1. The death receptor antagonist FLIP-L interacts with Trk and is necessary for neurite outgrowth induced by neurotrophins

    Journal of Neuroscience, Vol. 30, Núm. 17, pp. 6094-6105

2008

  1. A TrkB/EphrinA interaction controls retinal axon branching and synaptogenesis

    Journal of Neuroscience, Vol. 28, Núm. 48, pp. 12700-12712

2005

  1. The single AmphiTrk receptor highlights increased complexity of neurotrophin signalling in vertebrates and suggests an early role in developing sensory neuroepidermal cells

    Development, Vol. 132, Núm. 9, pp. 2191-2202

2004

  1. Trk is a calmodulin-binding protein: Implications for receptor processing

    Journal of Neurochemistry, Vol. 88, Núm. 2, pp. 422-433

2000

  1. Branched-chain amino acids inhibit proteolysis in rat skeletal muscle: Mechanisms involved

    Journal of Cellular Physiology, Vol. 184, Núm. 3, pp. 380-384

1999

  1. Ubiquitin gene expression is increased in human muscle undergoing neurogenic involvement

    Neurochemistry International, Vol. 34, Núm. 2, pp. 137-140

1998

  1. Different cytokines modulate ubiquitin gene expression in rat skeletal muscle

    Cancer Letters, Vol. 133, Núm. 1, pp. 83-87

  2. Protein turnover in skeletal muscle of tumour-bearing transgenic mice overexpressing the soluble TNF receptor-1

    Cancer Letters, Vol. 130, Núm. 1-2, pp. 19-27

  3. Role of TNF receptor 1 in protein turnover during cancer cachexia using gene knockout mice

    Molecular and Cellular Endocrinology, Vol. 142, Núm. 1-2, pp. 183-189

  4. Ubiquitin and proteasome gene expression is increased in skeletal muscle of slim AIDS patients.

    International journal of molecular medicine, Vol. 2, Núm. 1, pp. 69-73

1997

  1. Comparative effects of β2-adrenergic agonists on muscle waste associated with tumour growth

    Cancer Letters, Vol. 115, Núm. 1, pp. 113-118

  2. Lipid metabolism in tumour-bearing mice: Studies with knockout mice for tumour necrosis factor receptor 1 protein

    Molecular and Cellular Endocrinology, Vol. 132, Núm. 1-2, pp. 93-99

  3. TNF can directly induce the expression of ubiquitin-dependent proteolytic system in rat soleus muscles

    Biochemical and Biophysical Research Communications, Vol. 230, Núm. 2, pp. 238-241

1996

  1. Anti-TNF treatment reverts increased muscle ubiquitin gene expression in tumour-bearing rats

    Biochemical and Biophysical Research Communications, Vol. 221, Núm. 3, pp. 653-655

  2. Muscle hypercatabolism during cancer cachexia is not reversed by the glucocorticoid receptor antagonist RU38486

    Cancer Letters, Vol. 99, Núm. 1, pp. 7-14