Publicaciones en colaboración con investigadores/as de Instituto de Investigación Biomédica de Lleida (27)

2024

  1. Sprouty1 is a broad mediator of cellular senescence

    Cell Death and Disease, Vol. 15, Núm. 4

2023

  1. In Vivo Intra-Uterine Delivery of TAT-Fused Cre Recombinase and CRISPR/Cas9 Editing System in Mice Unveil Histopathology of Pten/p53-Deficient Endometrial Cancers

    Advanced Science, Vol. 10, Núm. 32

  2. Metformin exhibits antineoplastic effects on Pten-deficient endometrial cancer by interfering with TGF-β and p38/ERK MAPK signalling

    Biomedicine and Pharmacotherapy, Vol. 168

  3. Transient and DNA-free in vivo CRISPR/Cas9 genome editing for flexible modeling of endometrial carcinogenesis

    Cancer Communications

2022

  1. A dominant negative mutation uncovers cooperative control of caudal Wolffian duct development by Sprouty genes

    Cellular and Molecular Life Sciences, Vol. 79, Núm. 10

  2. Lack of extracellular matrix switches TGF-β induced apoptosis of endometrial cells to epithelial to mesenchymal transition

    Scientific Reports, Vol. 12, Núm. 1

2021

  1. Endometrial pten deficiency leads to smad2/3 nuclear translocation

    Cancers, Vol. 13, Núm. 19

2019

  1. Barley β-glucan accelerates wound healing by favoring migration versus proliferation of human dermal fibroblasts

    Carbohydrate Polymers, Vol. 210, pp. 389-398

  2. Sprouty1 controls genitourinary development via its N-terminal tyrosine

    Journal of the American Society of Nephrology, Vol. 30, Núm. 8, pp. 1398-1411

2017

  1. A Smad3-PTEN regulatory loop controls proliferation and apoptotic responses to TGF-β in mouse endometrium

    Cell Death and Differentiation, Vol. 24, Núm. 8, pp. 1443-1458

  2. Autophagy orchestrates adaptive responses to targeted therapy in endometrial cancer

    Autophagy, Vol. 13, Núm. 3, pp. 608-624

  3. Palbociclib has antitumour effects on Pten-deficient endometrial neoplasias

    Journal of Pathology, Vol. 242, Núm. 2, pp. 152-164

2016

  1. 7-dehydrocholesterol efficiently supports Ret signaling in a mouse model of Smith-Opitz-Lemli syndrome

    Scientific Reports, Vol. 6

  2. Sprouty1 haploinsufficiency accelerates pheochromocytoma development in Pten+/-mice

    Endocrine-Related Cancer

  3. Vascular Calcification Induced by Chronic Kidney Disease Is Mediated by an Increase of 1α-Hydroxylase Expression in Vascular Smooth Muscle Cells

    Journal of Bone and Mineral Research, Vol. 31, Núm. 10, pp. 1865-1876

2014

  1. Sprouty1 induces a senescence-associated secretory phenotype by regulating NFκB activity: Implications for tumorigenesis

    Cell Death and Differentiation, Vol. 21, Núm. 2, pp. 333-343

2013

  1. Differential gene expression of medullary thyroid carcinoma reveals specific markers associated with genetic conditions

    American Journal of Pathology, Vol. 182, Núm. 2, pp. 350-362

2012

  1. Sprouty1 is a candidate tumor-suppressor gene in medullary thyroid carcinoma

    Oncogene, Vol. 31, Núm. 35, pp. 3961-3972

2011

  1. KSR1 is overexpressed in endometrial carcinoma and regulates proliferation and TRAIL-induced apoptosis by modulating FLIP levels

    American Journal of Pathology, Vol. 178, Núm. 4, pp. 1529-1543

  2. Nuclear factor-B2/p100 promotes endometrial carcinoma cell survival under hypoxia in a HIF-1α independent manner

    Laboratory Investigation, Vol. 91, Núm. 6, pp. 859-871