Publicaciones en las que colabora con JOAQUIM EGEA NAVARRO (19)

2024

  1. Sprouty1 is a broad mediator of cellular senescence

    Cell Death and Disease, Vol. 15, Núm. 4

2023

  1. In Vivo Intra-Uterine Delivery of TAT-Fused Cre Recombinase and CRISPR/Cas9 Editing System in Mice Unveil Histopathology of Pten/p53-Deficient Endometrial Cancers

    Advanced Science, Vol. 10, Núm. 32

  2. Metformin exhibits antineoplastic effects on Pten-deficient endometrial cancer by interfering with TGF-β and p38/ERK MAPK signalling

    Biomedicine and Pharmacotherapy, Vol. 168

  3. Transient and DNA-free in vivo CRISPR/Cas9 genome editing for flexible modeling of endometrial carcinogenesis

    Cancer Communications

2022

  1. A dominant negative mutation uncovers cooperative control of caudal Wolffian duct development by Sprouty genes

    Cellular and Molecular Life Sciences, Vol. 79, Núm. 10

  2. Lack of extracellular matrix switches TGF-β induced apoptosis of endometrial cells to epithelial to mesenchymal transition

    Scientific Reports, Vol. 12, Núm. 1

2021

  1. Endometrial pten deficiency leads to smad2/3 nuclear translocation

    Cancers, Vol. 13, Núm. 19

2019

  1. Sprouty1 controls genitourinary development via its N-terminal tyrosine

    Journal of the American Society of Nephrology, Vol. 30, Núm. 8, pp. 1398-1411

2018

  1. Metabolomic Estimation of the Diagnosis and Onset Time of Permanent and Transient Cerebral Ischemia

    Molecular Neurobiology, Vol. 55, Núm. 7, pp. 6193-6200

  2. ProBDNF is modified by advanced glycation end products in Alzheimer's disease and causes neuronal apoptosis by inducing p75 neurotrophin receptor processing

    Molecular Brain, Vol. 11, Núm. 1

2004

  1. Trk is a calmodulin-binding protein: Implications for receptor processing

    Journal of Neurochemistry, Vol. 88, Núm. 2, pp. 422-433

2001

  1. Cytokines promote motoneuron survival through the janus kinase-dependent activation of the phosphatidylinositol 3-kinase pathway

    Molecular and Cellular Neuroscience, Vol. 18, Núm. 6, pp. 619-631

  2. Neuronal survival induced by neurotrophins requires calmodulin

    Journal of Cell Biology, Vol. 154, Núm. 3, pp. 585-597

2000

  1. Ca2+ and calmodulin regulate neuronal survival throughout the modulation of the intracellular signaling pathways activated by receptors for neurotrophic factors

    Methods and Findings in Experimental and Clinical Pharmacology

  2. Nerve growth factor activation of the extracellular signal-regulated kinase pathway is modulated by Ca2+ and calmodulin

    Molecular and Cellular Biology, Vol. 20, Núm. 6, pp. 1931-1946

1999

  1. Activation of phosphatidylinositol 3-kinase, but not extracellular- regulated kinases, is necessary to mediate brain-derived neurotrophic factor- induced motoneuron survival

    Journal of Neurochemistry, Vol. 73, Núm. 2, pp. 521-531

  2. Receptors of the glial cell line-derived neurotrophic factor family of neurotrophic factors signal cell survival through the phosphatidylinositol 3- kinase pathway in spinal cord motoneurons

    Journal of Neuroscience, Vol. 19, Núm. 21, pp. 9160-9169

1998

  1. Calmodulin is involved in membrane depolarization-mediated survival of motoneurons by phosphatidylinositol-3 kinase- and MAPK-independent pathways

    Journal of Neuroscience, Vol. 18, Núm. 4, pp. 1230-1239

  2. Development of survival responsiveness to brain-derived neurotrophic factor, neurotrophin 3 and neurotrophin 4/5, but not to nerve growth factor, in cultured motoneurons from chick embryo spinal cord

    Journal of Neuroscience, Vol. 18, Núm. 19, pp. 7903-7911