- Institut de Recerca Biomèdica de Lleida (IRB-Lleida)
Instituto de investigación
![Foto de Joan Xavier](/img/nophoto.png)
Joan Xavier
Comella Carnicé
Publicaciones en las que colabora con Joan Xavier Comella Carnicé (54)
2021
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Intracellular pathways involved in cell survival are deregulated in mouse and human spinal muscular atrophy motoneurons
Neurobiology of Disease, Vol. 155
2020
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SIVA-1 regulates apoptosis and synaptic function by modulating XIAP interaction with the death receptor antagonist FAIM-L
Cell Death and Disease, Vol. 11, Núm. 2
2019
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Genome Wide Meta-Analysis identifies common genetic signatures shared by heart function and Alzheimer’s disease
Scientific Reports, Vol. 9, Núm. 1
2014
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Histone deacetylase inhibitors promote glioma cell death by G2 checkpoint abrogation leading to mitotic catastrophe
Cell Death and Disease, Vol. 5, Núm. 10
2013
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A pathway involving HDAC5, cFLIP and caspases regulates expression of the splicing regulator polypyrimidine tract binding protein in the heart
Journal of Cell Science, Vol. 126, Núm. 7, pp. 1682-1691
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FAIM-L is an IAP-binding protein that inhibits XIAP ubiquitinylation and protects from fas-induced apoptosis
Journal of Neuroscience, Vol. 33, Núm. 49, pp. 19262-19275
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Neurobehavioral characterization of Endonuclease G knockout mice reveals a new putative molecular player in the regulation of anxiety
Experimental Neurology, Vol. 247, pp. 122-129
2012
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Translation of Myocyte Enhancer Factor-2 is induced by hypertrophic stimuli in cardiomyocytes through a Calcineurin-dependent pathway
Journal of Molecular and Cellular Cardiology, Vol. 53, Núm. 4, pp. 578-587
2011
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A new model to study spinal muscular atrophy: Neurite degeneration and cell death is counteracted by BCL-XL Overexpression in motoneurons
Neurobiology of Disease, Vol. 42, Núm. 3, pp. 415-426
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Endog links Bnip3-induced mitochondrial damage and caspase-independent DNA fragmentation in ischemic cardiomyocytes
PLoS ONE, Vol. 6, Núm. 3
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Endonuclease G is a novel determinant of cardiac hypertrophy and mitochondrial function
Nature, Vol. 478, Núm. 7367, pp. 114-118
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Ubiquitination of TrkA by Nedd4-2 regulates receptor lysosomal targeting and mediates receptor signaling
Journal of Neurochemistry, Vol. 117, Núm. 3, pp. 479-493
2010
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The death receptor antagonist FLIP-L interacts with Trk and is necessary for neurite outgrowth induced by neurotrophins
Journal of Neuroscience, Vol. 30, Núm. 17, pp. 6094-6105
2009
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Polypyrimidine tract binding proteins (PTB) regulate the expression of apoptotic genes and susceptibility to caspase-dependent apoptosis in differentiating cardiomyocytes
Cell Death and Differentiation, Vol. 16, Núm. 11, pp. 1460-1468
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Specific vulnerability of mouse spinal cord motoneurons to membrane depolarization
Journal of Neurochemistry, Vol. 110, Núm. 6, pp. 1842-1854
2008
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A TrkB/EphrinA interaction controls retinal axon branching and synaptogenesis
Journal of Neuroscience, Vol. 28, Núm. 48, pp. 12700-12712
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An alternative view of apoptosis in heart development and disease
Cardiovascular Research
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Analysis of Ret knockin mice reveals a critical role for IKKs, but not PI 3-K, in neurotrophic factor-induced survival of sympathetic neurons
Cell Death and Differentiation, Vol. 15, Núm. 9, pp. 1510-1521
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BCL-X L regulates TNF-α-mediated cell death independently of NF-kB, FLIP and IAPs
Cell Research, Vol. 18, Núm. 10, pp. 1020-1036
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Neuroprotection by neurotrophic factors and membrane depolarization is regulated by calmodulin kinase IV
Journal of Biological Chemistry, Vol. 283, Núm. 7, pp. 4133-4144