Publications by the researcher in collaboration with Joan Xavier Comella Carnicé (54)

2021

  1. Intracellular pathways involved in cell survival are deregulated in mouse and human spinal muscular atrophy motoneurons

    Neurobiology of Disease, Vol. 155

2020

  1. SIVA-1 regulates apoptosis and synaptic function by modulating XIAP interaction with the death receptor antagonist FAIM-L

    Cell Death and Disease, Vol. 11, Núm. 2

2019

  1. Genome Wide Meta-Analysis identifies common genetic signatures shared by heart function and Alzheimer’s disease

    Scientific Reports, Vol. 9, Núm. 1

2014

  1. Histone deacetylase inhibitors promote glioma cell death by G2 checkpoint abrogation leading to mitotic catastrophe

    Cell Death and Disease, Vol. 5, Núm. 10

2013

  1. A pathway involving HDAC5, cFLIP and caspases regulates expression of the splicing regulator polypyrimidine tract binding protein in the heart

    Journal of Cell Science, Vol. 126, Núm. 7, pp. 1682-1691

  2. FAIM-L is an IAP-binding protein that inhibits XIAP ubiquitinylation and protects from fas-induced apoptosis

    Journal of Neuroscience, Vol. 33, Núm. 49, pp. 19262-19275

  3. Neurobehavioral characterization of Endonuclease G knockout mice reveals a new putative molecular player in the regulation of anxiety

    Experimental Neurology, Vol. 247, pp. 122-129

2012

  1. Translation of Myocyte Enhancer Factor-2 is induced by hypertrophic stimuli in cardiomyocytes through a Calcineurin-dependent pathway

    Journal of Molecular and Cellular Cardiology, Vol. 53, Núm. 4, pp. 578-587

2011

  1. A new model to study spinal muscular atrophy: Neurite degeneration and cell death is counteracted by BCL-XL Overexpression in motoneurons

    Neurobiology of Disease, Vol. 42, Núm. 3, pp. 415-426

  2. Endog links Bnip3-induced mitochondrial damage and caspase-independent DNA fragmentation in ischemic cardiomyocytes

    PLoS ONE, Vol. 6, Núm. 3

  3. Endonuclease G is a novel determinant of cardiac hypertrophy and mitochondrial function

    Nature, Vol. 478, Núm. 7367, pp. 114-118

  4. Ubiquitination of TrkA by Nedd4-2 regulates receptor lysosomal targeting and mediates receptor signaling

    Journal of Neurochemistry, Vol. 117, Núm. 3, pp. 479-493

2010

  1. The death receptor antagonist FLIP-L interacts with Trk and is necessary for neurite outgrowth induced by neurotrophins

    Journal of Neuroscience, Vol. 30, Núm. 17, pp. 6094-6105

2009

  1. Polypyrimidine tract binding proteins (PTB) regulate the expression of apoptotic genes and susceptibility to caspase-dependent apoptosis in differentiating cardiomyocytes

    Cell Death and Differentiation, Vol. 16, Núm. 11, pp. 1460-1468

  2. Specific vulnerability of mouse spinal cord motoneurons to membrane depolarization

    Journal of Neurochemistry, Vol. 110, Núm. 6, pp. 1842-1854

2008

  1. A TrkB/EphrinA interaction controls retinal axon branching and synaptogenesis

    Journal of Neuroscience, Vol. 28, Núm. 48, pp. 12700-12712

  2. An alternative view of apoptosis in heart development and disease

    Cardiovascular Research

  3. Analysis of Ret knockin mice reveals a critical role for IKKs, but not PI 3-K, in neurotrophic factor-induced survival of sympathetic neurons

    Cell Death and Differentiation, Vol. 15, Núm. 9, pp. 1510-1521

  4. BCL-X L regulates TNF-α-mediated cell death independently of NF-kB, FLIP and IAPs

    Cell Research, Vol. 18, Núm. 10, pp. 1020-1036

  5. Neuroprotection by neurotrophic factors and membrane depolarization is regulated by calmodulin kinase IV

    Journal of Biological Chemistry, Vol. 283, Núm. 7, pp. 4133-4144