Publicaciones en las que colabora con Gustavo Barja de Quiroga Losada (66)

2023

  1. Introduction to special issue on ‘physiological and evolutionary mechanisms of aging’

    Experimental Gerontology

  2. Mitochondrial ROS production, oxidative stress and aging within and between species: Evidences and recent advances on this aging effector

    Experimental Gerontology, Vol. 174

  3. Phenotypic molecular features of long-lived animal species

    Free Radical Biology and Medicine, Vol. 208, pp. 728-747

  4. Programmed versus non-programmed evolution of aging. What is the evidence?

    Experimental Gerontology, Vol. 175

  5. Whole organism aging: Parabiosis, inflammaging, epigenetics, and peripheral and central aging clocks. The ARS of aging

    Experimental Gerontology, Vol. 174

2022

  1. Elovl2-Ablation Leads to Mitochondrial Membrane Fatty Acid Remodeling and Reduced Efficiency in Mouse Liver Mitochondria

    Nutrients, Vol. 14, Núm. 3

  2. Methionine Metabolism Is Down-Regulated in Heart of Long-Lived Mammals

    Biology, Vol. 11, Núm. 12

2021

  1. Is the NDUFV2 subunit of the hydrophilic complex I domain a key determinant of animal longevity?

    FEBS Journal

  2. mTORC1 is also involved in longevity between species

    Aging

2020

  1. Gene expression and regulatory factors of the mechanistic target of rapamycin (mTOR) complex 1 predict mammalian longevity

    GeroScience, Vol. 42, Núm. 4, pp. 1157-1173

  2. Low abundance of NDUFV2 and NDUFS4 subunits of the hydrophilic complex I domain and VDAC1 predicts mammalian longevity

    Redox Biology, Vol. 34

2016

  1. Rapamycin reverses age-related increases in mitochondrial ROS production at complex I, oxidative stress, accumulation of mtDNA fragments inside nuclear DNA, and lipofuscin level, and increases autophagy, in the liver of middle-aged mice

    Experimental Gerontology, Vol. 83, pp. 130-138

2015

  1. Cysteine dietary supplementation reverses the decrease in mitochondrial ROS production at complex I induced by methionine restriction

    Journal of Bioenergetics and Biomembranes, Vol. 47, Núm. 3, pp. 199-208

  2. Long lifespans have evolved with long and monounsaturated fatty acids in birds

    Evolution, Vol. 69, Núm. 10, pp. 2776-2784

2014

  1. Independent and additive effects of atenolol and methionine restriction on lowering rat heart mitochondria oxidative stress

    Journal of Bioenergetics and Biomembranes, Vol. 46, Núm. 3, pp. 159-172

  2. Lifelong treatment with atenolol decreases membrane fatty acid unsaturation and oxidative stress in heart and skeletal muscle mitochondria and improves immunity and behavior, without changing mice longevity

    Aging Cell, Vol. 13, Núm. 3, pp. 551-560

2013

  1. An evolutionary comparative view for longevity-related oxidative stress resistance mechanisms in animal species

    Natural Selection: Biological Processes, Theory and Role in Evolution (Nova Science Publishers, Inc.), pp. 145-174

  2. Formation of S-(carboxymethyl)-cysteine in rat liver mitochondrial proteins: Effects of caloric and methionine restriction

    Amino Acids, Vol. 44, Núm. 2, pp. 361-371

  3. La suplementación de larga duración con atenolol convierte la insaturación de las membranas mitocondriales de músculo esquelético y de corazón de ratón en la característica de mamíferos de supervivencia diez veces mayor. Efectos sobre la longevidad media y máxima

    Anales de la Real Academia Nacional de Farmacia, Vol. 79, Núm. 2, pp. 253-273

  4. Membrane lipid unsaturation as physiological adaptation to animal longevity

    Frontiers in Physiology, Vol. 4 DEC